How did Gbold know so much about all this stuff? Did he have some qualifications in biology/biochem? Or was he just completely self taught since he got ill? He should have pursued a career in this field because he is seriously smart
My criticism about TEI/ARL is that they don't really show us any explanations about their work or theories and I think they should it would be helpful, not sure why they have to keep it such a secret, Gbold said TEIs model is based on 10000 dimensions
Helen: "yes, this is what TEI and ARL do for slow oxidizers, they chelate copper . Copper lowers vitamin D3 levels and causes calcium to get out of the cell.
Since calcium goes out of the cell= this lead to lower protein kinase.= which lead to lower ionized calcium and also lower MHC expression. since protein kinase upregulates MHC. and vitamin D increases protein kinase by simply increasing calcium. which then upregulate MHC
I think this mechanim is created to control the bacterial infection IMHO. I might not be right.
I mean you talk about upregulating the MHC, but it is ionized calcium which upregulates it , not vitamin D. This is why they use calcium chloride which increases Ionized form of calcium. And actually regular calcium or high calcium diet will decrease ionized calcium. vitamin D increases calcium and phoshorus, thus if you give vitamin D with high phosphorus diet but low calcium diet, you will increase ionized calcium.
If you give vitamin D with high calcium diet, you will decrease ionized calcium.
this is why you talk about aspirin, If you give aspirin with calcium. You will increase ionized calcium, since it is similar to calcium chloride.
If you just give simple calcium , this will decrease ionized calcium.
IMO , this is how vitamin D works. vitamin D without calcium increases phosphorus, which increases ionized calcium, Ionized calcium increases MHC.
Phorphorus is needed to activate all vitamin Bs. This is why if there is a big pressure on glutathione enzymes, you run low on phosphorus and bs. And phosphorus is low on the hairtest since it is being used and calcium is high.
TEI just give phosphorus to slow oxidizers and B vitamins, with regular not high doses of vitamin D. This way you get active Bs, like B1 and B2 etc. and active b6.
Without this you cant take zinc. Since zinc will put pressure on already low vitamin B2 and phosphorus, since zinc turns vitamin B6 into an active form, and this makes niacin .turning of vitamin b6 into it its active form requires, b2 phosphorus magnesium and zinc. So to have NADPH you need zinc. and without NADPH you cant recycle glutathione.
Zinc alone will lower B2 levels and will cause lower ionized calcium. So slow oxidizers if they take zinc alone often see their calcium levels GO UP. Potassium goes up just because zinc increases CA and converts CO2 into bicarbonate. SO to take zinc alone is very very bad for a slow oxidizer.
MattyB , why dont look into those programs of TEI... those guys know all of those things and knew them for yeas, and all interactions. The current research is like 20 years behind.
People who try to create protocols end up creating their protocols. |But it is so hard to create one protocol for everyone. Why not just follow TEI.
Look at this document from TEI, they had this in 1989. and tested all of this for ages.
http://traceelements.com/Docs/News Nov-Dec 89.pdf
this is from 1989 .
Slow oxidizer does not have ionized calcium. .
Sometimes when I understand more and more of the interactions and I go, holy shit, TEiI already knew it. It is still amazing to me that people the current research is done on such a low levels. Like they study vitamin D< or study vitamins C, or study vitamin E/ but they dont study all interactions at once."